4 edition of Biochemistry and pharmacology of myocardial hypertrophy, hypoxia, and infarction found in the catalog.
Biochemistry and pharmacology of myocardial hypertrophy, hypoxia, and infarction
International Study Group for Research in Cardiac Metabolism.
Includes bibliographies and index.
|Statement||jointly held by the European and American research groups ; sponsored by Gesellschaft für Strahlen- und Umweltforschung mbH., Munich] ; edited by Peter Harris, Richard J. Bing, and Albrecht Fleckenstein.|
|Series||Recent advances in studies on cardiac structure and metabolism ;, v. 7|
|Contributions||Harris, Peter, 1923-, Bing, Richard J., 1909-, Fleckenstein, Albrecht., Gesellschaft für Strahlen- und Umweltforschung.|
|LC Classifications||RC667 .R4 vol. 7, RC685.H9 .R4 vol. 7|
|The Physical Object|
|Pagination||xi, 491 p. :|
|Number of Pages||491|
|LC Control Number||75043711|
to explore the effects of chronic systemic hypoxia on myocardial structure and morphology. In addition, the goal of present study is to develop a hypoxia-induced heart failure model in rats. Get this from a library! Cardiac glycosides biochemistry, pharmacology, clinical relevance. [Erland Erdmann; K Greeff; J C Skou;] -- The bicentenary of William Withering's now famous medical report entitled "An Account of the Foxglove and Some of its Medical Uses" has given us the occasion to analyze the state of the art. Cardiac.
Myocardial infarction ("heart attack") is the irreversible damage of myocardial tissue caused by prolonged ischemia and hypoxia. This most commonly occurs when a coronary artery becomes occluded following the rupture of an atherosclerotic plaque, which then leads to the formation of a blood clot (coronary thrombosis). Reviews of Physiology, Biochemistry and Pharmacology, Vol. by. Reviews of Physiology, Biochemistry and Pharmacology (Book ) Thanks for Sharing! You submitted the following rating and review. We'll publish them on our site once we've reviewed : Springer International Publishing.
Myocardial infarction after coronary ligation 3. Occlusion of coronary artery in anesthetized dogs 4. Acute ischemia by injection of microspheres in dogs 5. Influence on myocardial preconditioning Ex vivo methods 1. Plastic casts from coronary vasculature bed MODELS FOR SCREENING OF MYOCARDIAL INFARCTION ISOLATED ORGANS 1. Sodium/hydrogen exchange (NHE) inhibitors show promise as potential therapeutic agents for the treatment of heart failure, but it is not known whether they can reverse the maladaptive remodeling th Cited by:
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Get this from a library. Biochemistry and pharmacology of myocardial hypertrophy, hypoxia, and infarction: [proceedings of the sixth annual meeting of the International Study Group for Research in Cardiac Metabolism, SeptemberFreiburg, Germany.
[Peter Harris; Richard J Bing; Albrecht Fleckenstein; International Study Group for Research in Cardiac Metabolism. The Pathophysiology and Pharmacotherapy of Myocardial Infarction deals with the advances in the pathophysiology and pharmacotherapy of acute myocardial infarction and related complications.
This book covers two major consequences—cardiac muscle dysfunction and cardiac electrical Edition: 1. Biochemistry and pharmacology of myocardial hypertrophy Biochemistry and pharmacology of myocardial hypertrophy, hypoxia, and infarction/ edited by Peter Harris, Richard J. Bing, and Albrecht Fleckenstein.
Country of Publication: United States Publisher: Baltimore: University Park Press, c In recent years, cardiac metabolism has concerned itself primarily with the two conditions of interest to the clinician: myocardial infarction and myocardial failure. In both fields, disturbances in Author: R.
Bing, J. Sarma, R. Fischer, Y. Maruyama, R. Weishaar. Three experimental models were used: (i) 3–4 months’ old Wistar rats with normal blood pressure served as controls; (ii) hyperthyroid Wistar rats of 3–4 months of age were studied after two weeks of triiodothyronine injections (50 µg daily); (iii) rats of the same age after 2–4 weeks of hypobaric hypoxia which leads to hypertrophy of Cited by: 2.
Pathological cardiac hypertrophy. Cardiac hypertrophy is classified as pathological hypertrophy when it is associated with cardiac dysfunction (Shimizu and Minamino ).Pathological cardiac hypertrophy generally occurs in the presence of cardiovascular disease (CVD) (Tham et al.
), and it can ultimately contribute to the development of heart failure (Weeks and McMullen ).Cited by: 1. In acute myocardial infarction, the shock syndrome is associated with loss of hypoxia percent or more of left ventricular myocardium.
Since in-hospital prognosis in acute myocardial infarction is closely related to extent of left ventricular damage, estimation of the latter would be helpful in applying the most appropriate forms of therapy in these Cited by: The most common cause of replacement fibrosis is scarring from myocardial infarction.
Other conditions associated with replacement fibrosis include hypertrophic cardiomyopathy, eds. Biochemistry and pharmacology of myocardial hypertrophy, hypoxia, and infarction. Baltimore, Md: University Park Press, ; 99–Cited by: has increased during the last few decades.
Cardiac hypertrophy, a disease associated with the myocardium, is characterized by thickening of ventricle wall of heart and consequent reduction in the contracting ability of heart to pump the blood. Cardiac hypertrophy has been divided into two types, i.e.
physiological and pathological Size: KB. Myocardial infarction ("heart attack") is the irreversible damage of myocardial tissue caused by prolonged ischemia and hypoxia. This most commonly occurs when a coronary artery becomes occluded following the rupture of an atherosclerotic plaque, which then leads to the formation of a blood clot (coronary thrombosis).
This book summarizes present knowledge of different mechanisms involved in the development of positive and negative consequences of cardiac adaptation. Particular attention is paid to the still underestimated adaptive cardiac responses during development, to adaptation to the frequently occurring pressure and volume overload as well as to cardiac changes, induced by enduring exercise and.
The study is based on the experimental model of myocardial hypertrophy in rats. Male Sprague-Dawley adult rats were exposed to acute hypoxia in a hypo Cited by: 1. Kumar S, Wang G, Liu W, Ding W, Dong M, Zheng N, et al. Hypoxia-induced mitogenic factor promotes cardiac hypertrophy via calcium-dependent and hypoxia-inducible factor-1α mechanisms.
Hypertension Crossref, Medline, ISI, Google by: 1. Myocardial infarction is a major cause of morbidity and mortality worldwide. However, the methodological development of a spatiotemporally controllable investigation of the damage events in myocardial infarction remains challengeable.
In the present study, we describe a micropillar array-aided tissue interface mimicking microfluidic device for the dynamic study of hypoxia-induced myocardial Cited by: Myocardial infarction, or "heart attack," is irreversible damage to myocardial tissues caused by prolonged ischemia/hypoxia and by reperfusion-induced injury.
The damaged tissue is initially composed of a necrotic core surrounded by a marginal (or border) zone that can recover or. The molecular basis of myocardial hypertrophy and heart failure Oliver Ritter1 and Ludwig Neyses2 1Department of Medicine, University of Wuerzburg, Josef Schneider Str.
2, Wuerzburg, Germany 2Manchester Heart Center, University of Manchester, Manchester Royal Inﬁrmary, Oxford Road, Manchester, UK M13 9WL Heart failure (HF) is the inability of the heart to cope. INTRODUCTION. Ischemia occurs when blood flow to the myocardium is reduced ().Ischemia of prolonged duration induces myocardial infarction (MI), and MI is a common cause of heart failure ().Ischemic cardiomyopathy is the most common cause of heart failure and can arise from remodeling after an acute ST segment elevation myocardial infarction (STEMI) from multiple small Cited by: Zentilin, L.
et al. Cardiomyocyte VEGFR-1 activation by VEGF-B induces compensatory hypertrophy and preserves cardiac function after myocardial infarction. FASEB J. 24. The bicentenary of William Withering's now famous medical report entitled "An Account of the Foxglove and Some of its Medical Uses" has given us the occasion to analyze the state of the art.
Cardiac glycosides in are considered tobe the basis for medical treatment of myocardial failure. Ischemic heart disease, especially myocardial infarction (MI), is the leading cause of death worldwide.
Apoptotic mechanisms are thought to play a significant role in cardiomyocyte death after MI. Increased production of heat shock proteins (Hsp) in cardiomyocytes is a normal response to promote tolerance and to reduce cell damage.
Hsp27 is considered to be a therapeutic option for the. The data for cTn were so compelling that in a global task force comprising representatives from the European Society of Cardiology (ESC) and the American College of Cardiology (ACC) was convened to redefine the diagnosis of MI based on cTn measurements.1 Recently, the National Academy of Clinical Biochemistry (NACB) has published updated.
Pharmacological Control of Heart and Circulation, Volume 1, documents the proceedings of the 3rd Congress of the Hungarian Pharmacological Society held in Budapest, Because the papers published in this volume cover diverse areas of the cardiovascular field, they had to be arranged into several smaller Edition: 1.Pathophysiology of Myocardial Infarction.
Volume 5. Issue 4. October Infarct healing is intertwined with geometric remodeling of the chamber, characterized by dilation, hypertrophy of viable segments, and progressive dysfunction.
This review manuscript describes the molecular signals and cellular effectors implicated in injury, repair Cited by: